By M. V. Singer, D. Brenner
Read or Download Alcohol And the Gastrointestinal Tract: Special Issue: Digestive Diseases 2005 PDF
Similar diseases books
A concise and sensible, but commprehensive textbook masking the analysis and administration of acute and protracted discomfort. The booklet addresses the physiological and pharmacological foundation of soreness mechanisms, offers differential diagnoses, and develops rationales and techniques of remedy.
This can be a 3-in-1 reference ebook. It offers a whole clinical dictionary overlaying thousands of phrases and expressions when it comes to sympathetic dystrophy. It additionally supplies vast lists of bibliographic citations. ultimately, it presents details to clients on how one can replace their wisdom utilizing numerous web assets.
Oxidative rigidity and irritation underpin so much illnesses; their mechanisms are inextricably associated. for instance, continual irritation is linked to oxidation, anti inflammatory cascades are associated with lowered oxidation, elevated oxidative rigidity triggers irritation and redox stability inhibits the inflammatory mobile reaction.
- Acute Bronchitis - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
- The Black Death: The Great Mortality of 1348-1350: A Brief History with Documents (Bedford Series in History & Culture)
- Virus Diseases in Laboratory and Captive Animals
- Autoantibodies in Neurological Diseases
- Inflammatory Bowel Diseases 1990: Proceedings of the Third International Symposium on Inflammatory Bowel Diseases, Jerusalem, September 10–13, 1989
- Diseases of the Thyroid
Additional info for Alcohol And the Gastrointestinal Tract: Special Issue: Digestive Diseases 2005
Slightly increased number of mitotic ﬁgures. Maier et al.  also looked at the effects of chronic ethanol consumption on the oral mucosa in a rat animal model. They observed a signiﬁcant increase in the size of the basal cell layer and basal cell nuclei of oral epithelium and an increase in the percentage of cells in the S-phase of the cell cycle. The mean epithelial thickness was reduced. They suggested that the chronic ethanol consumption caused oral mucosal atrophy with associated hyper-regeneration, which in turn may result in an enhanced susceptibility of the mucosal epithelium towards chemical carcinogens.
Alcoholics, by deﬁnition, are addicted to excessive use of alcohol. Their cancer mortality and incidence rates have been studied in several countries and compared with the prevailing rates in the general population . A 3- to 6-fold increased risk of head and neck cancer compared with the general population was reported for these alcoholics, the risk tended to be higher for oral and pharyngeal cancers than for laryngeal cancer. Cohort studies, looking at alcoholics in Norway, Sweden, Canada and the USA, have found an increased risk for oral cancer compared with the general population [7, 25].
Moreover, in vivo salivary acetaldehyde levels correlate very signiﬁcantly with the levels that are produced in vitro. This offers the opportunity to use the in vitro salivary test as a tool to investigate possible variables which might inﬂuence salivary acetaldehyde production. Salivary acetaldehyde levels after ethanol intake strikingly exceed those known to be derived from endogenous metabolism of ethanol . Via normal distribution and evaporation, salivary acetaldehyde may reach all target tissues of the upper aerodigestive tract.
Alcohol And the Gastrointestinal Tract: Special Issue: Digestive Diseases 2005 by M. V. Singer, D. Brenner